Nuclear Receptor Signaling.js

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	"label": "Nuclear Receptor Signaling",
	"creator": "Aurimas Vinckevicius",
	"target": "^https?://[^/]*nursa.org/(article|nrs|abstract)",
	"minVersion": "3.0",
	"maxVersion": "",
	"priority": 100,
	"inRepository": true,
	"translatorType": 4,
	"browserSupport": "gcsibv",
	"lastUpdated": "2014-03-03 22:02:54"
}

/*
   Nuclear Receptor Signaling Translator
   Copyright (C) 2012 Aurimas Vinckevicius

   This program is free software: you can redistribute it and/or modify
   it under the terms of the GNU Affero General Public License as published by
   the Free Software Foundation, either version 3 of the License, or
   (at your option) any later version.

   This program is distributed in the hope that it will be useful,
   but WITHOUT ANY WARRANTY; without even the implied warranty of
   MERCHANTABILITY or FITNESS FOR A PARTICULAR PURPOSE.  See the
   GNU Affero General Public License for more details.

   You should have received a copy of the GNU Affero General Public License
   along with this program.  If not, see <http://www.gnu.org/licenses/>.
 */


/*
 Translator for Nuclear Receptor Signaling journal. Example URLs:
 	Multiple:
 		http://www.nursa.org/nrs.cfm?detail=Perspectives&journalVolume=9
 		http://www.nursa.org/nrs.cfm?detail=Reviews&journalVolume=9
 		http://www.nursa.org/nrs.cfm?detail=Most%20Viewed&journalVolume=9
 		http://www.nursa.org/nrs.cfm?detail=Methods&journalVolume=9
 	Journal Article:
 		http://www.nursa.org/abstract.cfm?doi=10.1621/nrs.07008
 		http://www.nursa.org/article.cfm?doi=10.1621/nrs.09001
*/

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		var title, itemUrl;
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			title = Zotero.Utilities.xpathText(items[i], './i[text()="Nucl Recept Signal"]/preceding-sibling::node()', null, ' ');
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		item.publicationTitle = 'Nuclear Receptor Signaling';
		item.journalAbbreviation = 'Nucl. Recept. Signaling';

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}

/** BEGIN TEST CASES **/
var testCases = [
	{
		"type": "web",
		"url": "http://www.nursa.org/article.cfm?doi=10.1621/nrs.09002",
		"items": [
			{
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				"creators": [
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						"firstName": "Stephen",
						"lastName": "Safe",
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						"lastName": "Li",
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				"title": "NR4A orphan receptors and cancer",
				"publicationTitle": "Nuclear Receptor Signaling",
				"journalAbbreviation": "Nucl. Recept. Signaling",
				"date": "March 18, 2011",
				"volume": "9",
				"pages": "e002",
				"abstractNote": "NR4A orphan receptors are members of the nuclear receptor (NR) superfamily of transcription factors and include NR4A1 (Nur77, TR3, NGFI-B), NR4A2 (Nurr1), and NR4A3 (Nor1). NR4A receptors are immediate-early genes induced by multiple stimuli and modulate gene expression by interacting as monomers or homodimers to NGFI-B response elements (NBREs) and Nur-responsive elements (NuREs), respectively. NR4A1 and NR4A2 (but not NR4A3) also form heterodimers with the retinoic acid X receptor (RXR) that bind a DR5 motif, and there is evidence that NR4A1 can activate or deactivate gene expression in cancer cells through interactions with DNA-bound specificity protein 1(Sp1) transcription factor. NR4A receptors play important roles in cellular homeostasis and disease, and there is increasing evidence that they exhibit pro-oncogenic activity in most tumors and thereby represent novel targets for chemotherapeutic drugs. Many apoptosis-inducing drugs induce nuclear export of NR4A1 and activate apoptosis in cancer cell lines through formation of extranuclear complexes including a pro-apoptotic mitochondrial NR4A1-bcl-2 complex. 1,1-Bis(3'-indolyl)-1-(p-substituted phenyl)methane analogs exhibit structure-dependent activation or deactivation of nuclear NR4A1 to induce apoptosis, whereas cytosporone B and structural analogs activate both nuclear and extranuclear NR4A1-dependent pro-apoptotic pathways. The roles of NR4A2 and NR4A3 in carcinogenesis are less well-defined; however, there is evidence suggesting that NR4A receptors are important targets for development of new mechanism-based anticancer drugs.",
				"DOI": "10.1621/nrs.09002",
				"url": "http://www.nursa.org/article.cfm?doi=10.1621/nrs.09002",
				"ISSN": "1550-7629",
				"rights": "Copyright © 2011, Safe et al. This is an open-access article distributed under the terms of the Creative Commons Non-Commercial Attribution License, which permits unrestricted non-commercial use distribution and reproduction in any medium, provided the original work is properly cited.",
				"accessDate": "CURRENT_TIMESTAMP",
				"libraryCatalog": "Nuclear Receptor Signaling"
			}
		]
	}
]
/** END TEST CASES **/